Mcseatons Kennels

          VITAMIN C SUPPLEMENTS
THERE IS MUCH CONTROVERSY ABOUT GIVING EXTRA VITAMINS OR SUPPLEMENTS TO PUPPIES
THERE ARE MANY ARTICLES FOR IT AND AGAINST IT.
ROUGHLY ONE IN FOUR HUNTING TYPE DOGS BECOME VICTIMS OF HIP DYSPEPSIA. SOME OF THESE DOGS SUFFER A LIFETIME OF CRIPPLING PAIN. OTHERS ARE DESTROYED. BUT IT IS NOW A VIRTUAL CERTAINTY THAT IF YOU TAKE SOME PREVENTIVE MEASURES, YOUR PUP WILL NOT GET HIP DYSPEPSIA. IS THIS A STRONG STATEMENT? READ ON , AND JUDGE FOR YOURSELF.



          SCURVY AND HIP DYSPEPSIA
SAN JOSE, CALIFORNIA VETERINARIAN DR. WENDELL O. BELFIELD WAS VISITING A FRIEND. ON THE TABLE WAS A REPRINT COPY OF THE 1753 BOOK "A TREATISE ON SCURVY" BY CAPTAIN JAMES LIND, A SURGEON IN THE BRITISH ROYAL NAVY. WHILE WAITING FOR HIS FRIEND DR. BELFIELD BEGAN READING.
THE SYMPTOMS DESCRIBED WERE: BLEEDING GUMS, LOOSE TEETH, FOUL BREATH,NOSE BLEEDS,SWOLLEN EYE LIDS,BRITTLE BONES.....AND THEN CAME AN AUTOPSY REPORT MADE IN 1699 AT ST. LOUIS HOSPITAL, PARIS : "THE LIGAMENTS OF THE JOINTS WERE CORRODED AND LOOSE. INSTEAD OF FINDING IN THE CAVITIES OF THE JOINTS THE USUAL SWEET OIL MUCILAGE, THERE WAS ONLY A GREENISH LIQUID, WHICH BY ITS CAUSTIC QUALITY HAD CORRODED THE LIGAMENTS."
RECOGNITION FLASHED ACROSS DR. BELFIELDS MIND. THIS 300 YEAR OLD AUTOPSY OF A DEAD HUMAN BEING DESCRIBED HIP DYSPEPSIA IN DOGS. COULD CHRONIC HIP DYSPEPSIA BE A FORM OF SCURVY? A SIMPLE LACK OF VITAMIN C? 
NO, IT'S ACCEPTED THAT A DOGS BODY MAKES ITS OWN VITAMIN C. EVERYBODY KNOWS THAT. CANINE HIP DYSPEPSIA IS HEREDITARY. EVERYBODY KNOWS THAT TOO. BUT WHY DOES EVERYBODY KNOW THAT DOGS DON'T NEED EXTRA VITAMIN C? BECAUSE BACK IN THE 1930S, SOME DOGS IN TESTING LABORATORIES SUFFERED NO ILL EFFECTS WHEN DENIED ALL VITAMIN C IN THEIR DIETS.
WE CANT HELP BUT OBSERVE, HOWEVER, THAT VERY LITTLE HAPPENS IN THE CONTROLLED ENVIRONMENT OF AN EXPERIMENTAL LABORATORY THAT WOULD CAUSE DOGS TO NEED MORE C THAN ITS BODY CAN MANUFACTURE. IN THE REAL WORLD A DOG SLED RACER BROUGHT A BITCH TO DR. BELFIELD AFTER OTHER VETS IN THE SAN FRANCISCO AREA HAD FAILED TO HELP HER. SHE HAD A SWOLLEN TONGUE, BLEEDING GUMS, AND OFTEN FELL AND DRAGGED HERSELF ABOUT WITH GREAT DIFFICULTY.
DR. BELFIELD RECOGNIZED THE TROUBLE AS SCURVY, ALTHOUGH NOT THE TRUE CLINICAL SCURVY THAT BRINGS DEATH TO HUMANS. THE DOGS WAS MAKING ENOUGH C TO KEEP SCURVY ON A CHRONIC LEVEL. AFTER A MONTH ON ASORBIC ACID (ANOTHER NAME FOR VITAMIN C) THE BITCH COULD RUN AS LONG AND HARD AS ANY DOG ON THE CLIENTS TEAM. WHY DIDN'T OTHER MEMBERS OF THE RACING TEAM HAVE SCURVY? THEY HAD BEEN EATING THE SAME FOOD. THIS HAPPENED BECAUSE OF ANOTHER FACTOR  USUALLY IGNORED BY THOSE WHO BELIEVE DOGS NEVER NEED MORE C THAN THEIR BODIES MAKE. IN ONE GROUP OF 104 DOGS TESTED VITAMIN C LEVELS VARIED FROM .02 MILLIGRAMS PER CUBIC CENTIMETER OF BLOOD TO .84 MILLIGRAMS- A 42 FOLD VARIABLE. OBVIOUSLY, ONE DOGS BODY CAN BE STARVING FOR C WHILE ANOTHER DOG MAKES ENOUGH TO GET BY.
THIS VARIABILITY ALSO LEADS US TO THE HEREDITARY FACTOR. BREEDING PROGRAMS TO ELIMINATE CDH HAVE WORKED TO A CERTAIN DEGREE. FOR INSTANCE, THE WIRE HAIRED POINTING GRIFFIN BREEDER ELIMINATED HALF OF THE PROBLEM IN JUST TEN YEARS THROUGH SELECTIVE BREEDING. BUT NO BREEDING PROGRAM IS CLOSE TO BEING 100% EFFECTIVE. IF WE ASSUME DR. BELFIELD IS RIGHT IN BELIEVING THAT HIP DYSPEPSIA IS CONNECTED TO VITAMIN C DEFICIENCY, IT ALL BEGINS TO MAKE SENSE. THE VARIABLE ABILITY TO MAKE ASORBIC ACID IS INHERITABLE, WHILE SELECTING PARENTS FOR LACK OF HIP DYSPEPSIA THE HEREDITARY, WE MAY HAVE UNKNOWINGLY SELECTED  FOR THE REAL FACTOR.
WHAT INHIBITS CHD - GOOD VITAMIN C PRODUCTION
IT SEEMS CLEAR. HOWEVER, THAT ALL CHD IS NOT ENTIRELY GENETIC. SELECTIVE BREEDING HAS BEEN TOO INCONSISTENTLY SUCCESSFUL TO BELIEVE OTHERWISE. DR BELFIELD SAYS THAT HE CONSTANTLY SEES SOUND PARENTS. A SWEDISH DOCTOR WHO X-RAYED ARMY DOGS CONCLUDED THAT DYSPLASTIC DOGS PRODUCE ONLY TEN PERCENT MORE DYSPLASIC PUPS THAN NORMAL DOGS. BUT AGAIN IF WE ACCEPT DR. BELFIELDS VITAMIN C THEORY IT ALL MAKES SENSE.
VITAMIN C DOES 300 DIFFERENT JOBS IN THE BODIES OF ANIMALS, INCLUDING HUMANS. ONE OF THE MOST IMPORTANT IS COLLAGEN PRODUCTION. IF YOU THINK OF CELLS AS BRICKS, COLLAGEN WOULD BE THE MORTAR.WITHOUT ENOUGH COLLAGEN YOU CANT BUILD MUSCLE TISSUE. YOU CANT BUILD BONE EITHER. BECAUSE COLLAGEN FORMS THE HONEYCOMB HOLDING THE MINERALS IN PLACE IN BONES. FURTHERMORE, ITS POSSIBLE TO HAVE NOT ENOUGH VITAMIN C TO MANUFACTURE QUALITY COLLAGEN. WEAK COLLAGEN BUILDS WEAK MUSCLES AND BONES.
A SECOND FUNCTION OF ASORBIC ACID IS TO COPE WITH THE EFFECTS OF STRESS. IT DOES THIS BY NOURISHING THE ADRENAL GLANDS AND BY HELPING THE BODY PRODUCE ITS OWN CORTISONE. WHICH COMBATS HIS AMINES PRODUCED BY DIEING CELLS. A RAT CAN MULTIPLY ITS VITAMIN C OUTPUT TEN FOLD WHEN STRESSED. HUMANS CANT MAKE ANY AT ALL, MUCH LESS INCREASE IT. SO WE MUST EAT ALL THAT OUR BODIES NEED OR SUPPLEMENT OUR DIETS. APPARENTLY OUR ANCESTORS ATE HUGE AMOUNTS OF FRUIT CONTAINING VITAMIN C SO UNLIKE ANIMALS OUR LIVERS DIDN'T DEVELOP THE ABILITY TO TURN GLUCOSE INTI ASORBIC ACID. DOGS ARE POOR PRODUCERS. A GOAT CAN MAKE FIVE TIMES THE VITAMIN C PRODUCED BY A DOG OF EQUALIZES. A RAT, SMALL AS IT IS MAKES NEARLY FOUR TIMES THE VITAMIN C PRODUCED BY A FULL GROWN DOG.
I HATE THE TERM"CONVENTIONAL WISDOM." TOO OFTEN IT MEANS BELIEVING WHAT EVERYBODY ELSE BELIEVES, NO MATTER HOW FOOLISH, FOR FEAR OF BEING RIDICULED.BUT IN THIS CASE, THE DEFINITION IS ACCURATE. CONVENTIONAL WISDOM HAS IT THAT DOGS NEED NO MORE VITAMIN C THAN THEIR BODIES MAKE. DR BELFIELD ASKS WHY, IF THATS THE CASE , DO DOGS OR WILD CANINES , WHEN LEFT TO THEIR OWN DEVICES, ACT LIKE THEY NEED MORE? WHEN POSSIBLE, CANINES DO EAT FRUITS, BERRIES, AND VEGETATION CONTAINING VITAMIN C.
IN MY EXPERIENCE THERE ARE TWO TIMES WHEN DOGS EAT LARGE AMOUNTS OF GRASS AND OTHER AVAILABLE VEGETATION. ONE IS WHEN THEY RUN LONG AND HARD, AND THERE IS NO WATER TO BE FOUND. AT SUCH TIMES, THEY CHEW AND SWALLOW VEGETATION FOR THE MOISTURE.  THE OTHER TIME IS WHEN THEY HAVE BEEN KENNELED TOO LONG. EVEN SOME HARD-RUNNING BIRD DOGS AND HOUNDS OFTEN STOP TO EAT GREEN VEGETATION BEFORE TAKING OFF TO HUNT. WE'VE ALWAYS WONDERED WHY. WE EVEN THOUGHT UP SEVERAL EXPLANATIONS, BUT THEY DIDN'T RING TRUE. COULD THIS SIMPLY BE CAUSED BY THE NEED OF ADDITIONAL VITAMIN C?
KEEP IN MIND THERE IS NO EXTRA VITAMIN C IN MOST COMMERCIAL DOG FOODS. THATS NOT CRITICISM EITHER. ASORBIC ACID OXIDIZES RAPIDLY WHEN THE LID IS OFF THE CONTAINER. ALSO THE HIGH HEAT DURING THE EXTRUDING PROCESS WOULD PROBABLY DESTROY MOST OF THE VITAMIN C ADDED TO THE FOOD. ONE MANUFACTURER THAT DOES ADD  ASORBIC ACID MAKES NO CLAIMS FOR ITS BENEFITS, NOR IS THE AMOUNT SPECIFIED OR GUARANTEED TO BE IN THE BAG.
TO SUM UP SO FAR, OUR DOG IS A POOR PRODUCER OF VITAMIN C THERE IS NO EXTRA C IN HIS USUAL FOOD AND LIKE US HE NEEDS FAR MORE IN PROPORTION TO HIS SIZE THAN WE DO TO ACHIEVE NORMAL GROWTH. WHILE OUR GROWTH IS STRETCHED OUT OVER TWO DECADES, MOST OF THEIRS TAKES PLACE DURING THE FIRST YEAR. THE BIGGER THE BREED OR STRAIN, THE MORE RAPID THE GROWTH, AND THE GREATER THE DEMAND FOR ASORBIC ACID, THE GREATER THE INCIDENCE OF HIP DYSPEPSIA.
DR. BELFIELD SUGGESTS THAT WE SHOULD ALSO CONSIDER THE STRESS ASSAULT ON THE DOMESTICATED PUP. IT'S WEANED, SEPARATED FROM ITS MOTHER AND LITTER MATES, INVOLUNTARILY CARRIED TO A NEW AND UNFAMILIAR LOCATION, STUCK WITH NEEDLES, MILDLY POISONED TO ELIMINATE WORMS, AND POSSIBLY OPERATED ON TO REMOVE DEWCLAWS OR PART OF A TAIL. ALL OF THIS HAPPENS WHILE THE ANIMAL IS ALREADY STRESSED BY TEETHING AND PHENOMENAL GROWTH. THE GROWTH FACTOR INDUCES A VERY HIGH DEMAND FOR ASORBIC ACID THROUGH ADDITIONAL STRESS AND THE MASSIVE NEED FOR COLLAGEN.THE WILD CANINE PUP, BY CONTRAST, STAYS WITH ITS MOTHER, KEEPS ITS TAIL AND DEWCLAWS,SUFFERS NO EARLY SEPARATION, IS NOT HURT BY HYPODERMIC NEEDLES , AND HAS NOT BEEN BRED OVERLARGE BY FOOLISH HUMANS. DISEASES AND WORMS ARE ITS STRESSES. BUT THE WILD PUP DOES GET EXTRA VITAMIN C FROM THE LIVERS OF ANIMALS THE MOTHER KILLS. SOME GREEN VEGETATION, AND SOMETIMES FROM FRUITS AND BERRIES. THE DOMESTIC PUP GETS NONE BECAUSE WE HAVE DECIDED THAT HE DOESN'T NEED ANY.
OUR PUP-IN A CONDITION PERHAPS BORDERING ON SUB CLINICAL SCURVY - RUNS ,PLAYS,AND JUMPS WITH MUSSELS AND BONES WEAK FROM LACK OF QUALITY COLLAGEN. MUSCLE GROWTH MAY NOT BE KEEPING PACE WITH BONE GROWTH. THE PECTINEUS MUSCLES BECOME TAUT. SUDDENLY DURING ONE GREAT LEAP OR UNUSUAL JOLT, THE WEAK MUSCLES FAIL, AND THE BALLS OF THE LEG BONES ARE PULLED AWAY FROM THE HIP SOCKETS. THE LUBRICATING SYNOVIAL FLUID LEAKS OUT, AND THE BALLS GRATE IN THE SOCKETS. IN HOURS MAYBE LESS, THE DOG HAS HIP DYSPEPSIA. IT WILL NOT RECOVER.
          THE SOLUTION
DR. BELFIELD THOUGHT THAT THE SOLUTION WAS OBVIOUS. PREVENT HIP DYSPEPSIA BY SUPPLYING ENOUGH VITAMIN C. HE TRIED IT WITH SEVERAL LITTERS OF GERMAN SHEPHERD PUPS, A BREED WITH SERIOUS CHD PROBLEMS. THE PARENTS EITHER HAD HIP DYSPEPSIA THEM SELFS OR HAD ALREADY PRODUCED DYSPEPTIC PUPS.
THE FIRST BITCH HAD VERY BAD HIPS AND ACCORDING TO SEEING EYE DOG STANDARDS SHOULD HAVE BEEN NEUTERED TO PREVENT PREGNANCY. DR BELFIELD GAVE HER 2,000 MILLIGRAMS (2 GRAMS) OF C DAILY AS SOON AS SHE WAS PREGNANT. EIGHT PUPS WERE BORN, AND THEY WERE GIVEN 50 TO 100 MILLIGRAMS OF LIQUID C FROM BIRTH UNTIL WEANED. FROM THAT POINT TO FOUR MONTHS, 550 MILLIGRAMS OF POWDERED C WERE ADDED TO THEIR FOOD. THIS WAS INCREASED TO 1,000 MILLIGRAMS, THEN 2,000 UNTIL THE PUPS WERE 18 TO 24 MONTHS OLD. NONE OF THE PUPS WERE DYSPLASIC.
ANOTHER BITCH HAD BEEN BRED TWICE TO TWO DIFFERENT STUDS THAT WERE CERTIFIED FREE OF CHD. EACH TIME, HALF OF HER PUPS WERE DYSPLASIC. SHE WAS BRED AGAIN AND VITAMIN C WAS ADMINISTERED UNDER DR. BELFIELDS GUIDANCE. SHE PRODUCED ELEVEN PUPS WITH PERFECT HIPS. IN ALL EIGHT LITTER WERE HANDLED IN THIS MATTER OVER A FIVE YEAR PERIOD. WHEN X-RAYED AT TWO YEARS OF AGE, ALL PUPS WERE FREE OF CHD.
THE TESTING ENDED IN1976, AND AS FAR AS DR. BELFIELD WAS CONCERNED, THE CHD THREAT WAS DEFEATED. HE PUBLISHED A PAPER IN A PROFESSIONAL JOURNAL. WAS IT TRULY OVER? HARDLY. DR. BELFIELD HAD ATTACKED THE POPULAR GENETIC THEORY THAT EVERYONE "KNEW." INSTEAD OF SIMPLY TRYING THE TREATMENT AND FINDING OUT FOR THEM SELFS, MOST OF THE PROFESSIONALS RIDICULED DR. BELFIELD FOR NOT CONDUCTING A BETTER EXPERIMENT WITH DOUBLE BLINDS (SOME DOGS NOT GIVEN C). HE WAS EVEN CALLED A CROOK AND A CHARLATAN.
"THAT REALLY HURT" DR. BELFIELD " I'M JUST A LITTLE ONE MAN PRACTITIONER WHO LOOKED FOR BETTER WAYS WHEN CONVENTIONAL MEANS WEREN'T WORKING. PROOF IS UP TO THE UNIVERSITIES. I REALLY WANT TO HELP THESE ANIMALS, NOT WAIT AROUND FOR FURTHER PROOF WHILE THE DOGS SUFFER."
WHATEVER YOU DO, IF YOUR RAISING A LITTER, PROVIDE THE NEEDED VITAMIN C. IF YOU'RE BUYING A PUP, PUT IT ON THE C THE MOMENT YOU TAKE IT HOME. THERE'S NO SENSE RISKING HIP DYSPEPSIA JUST BECAUSE ABSOLUTE PROOF HASN'T YET BEEN ESTABLISHED OR SERIOUSLY ATTEMPTED.